FILE - In this Feb. 10, 2013 file photo, Chris Brown arrives at the 55th annual Grammy Awards, in Los Angeles. Brown on Wednesday Nov. 6, 2013, countersued a man who claimed the R&B singer injured him during a fight outside a recording studio earlier this year. Brown's suit seeks unspecified damages and claims Sha'keir Duarte punched and kicked him during the fight. (Photo by Jordan Strauss/Invision/AP, File)
LOS ANGELES (AP) — Chris Brown has countersued a man who accused the R&B star's entourage of attacking him outside a recording studio earlier this year.
The singer filed an assault and battery lawsuit Wednesday against Sha'keir Duarte, who claimed in an earlier suit that he was injured when a fight erupted between Brown and Frank Ocean's entourages in January outside a West Hollywood studio.
Duarte sued Brown in August and accused the singer of being the aggressor in the fight. Brown's countersuit however accuses Duarte of instigating the fight by pushing, kicking and punching the R&B singer and threatening to kill him.
Brown, 24, is seeking unspecified damages.
Duarte's attorney Joseph Porter III did not immediately return a phone message seeking comment.
No criminal charges were filed over the fight, but Brown may face criminal penalties after he was arrested last month in Washington, D.C. for allegedly punching a man outside a hotel.
Brown remains on probation for his 2009 attack on then-girlfriend Rihanna and is due for a hearing in Los Angeles on Nov. 20, during which the new case may be addressed. Brown spent a day and a half in custody and faces a misdemeanor battery charge over the incident.
The R&B singer entered rehab for anger management issues on Oct. 29.
UT Southwestern researchers identify how body clock affects inflammation
PUBLIC RELEASE DATE:
7-Nov-2013
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Contact: Deborah Wormser deborah.wormser@utsouthwestern.edu 214-648-3404 UT Southwestern Medical Center
DALLAS Nov. 7, 2013 UT Southwestern Medical Center researchers report that disrupting the light-dark cycle of mice increased their susceptibility to inflammatory disease, indicating that the production of a key immune cell is controlled by the body's circadian clock.
The study published in the Nov. 8 edition of Science identifies a previously hidden pathway by which the body's circadian clock controls the numbers of key inflammatory cells called interleukin-17-producing CD4+ T helper cells (TH17). The work could lead to new ways to rev up the body's immune response to infection or dampen that response in the case of autoimmune diseases in which the body attacks its own tissues, said senior author Dr. Lora Hooper, Professor of Immunology and Microbiology and a Howard Hughes Medical Institute (HHMI) Investigator.
Co-authors include Neuroscience Chair and HHMI Investigator Dr. Joseph Takahashi, whose discovery of the mouse and human clock genes led to a description of a conserved circadian clock mechanism in animals. The lead author is Xiaofei Yu, an Immunology student in the UT Southwestern Graduate School of Biomedical Sciences.
"Virtually all life forms on Earth undergo physiological and behavioral changes on a 24-hour daily, or circadian, cycle in accordance with the changes in natural light. Human beings are no exception. Many of our physiological processes, such as eating and sleeping, vary dramatically between day and night. Such processes are controlled by a group of proteins, collectively termed the 'circadian clock,' which function together in individual cells, capturing light cues from the visual and nervous systems and using these cues to regulate gene expression," explained Dr. Hooper, who holds appointments in the Center for the Genetics of Host Defense and the Cancer Immunobiology Center.
Although the circadian clock is known to regulate metabolism and sleep-wake cycles, little was known about whether the circadian clock also regulates the immune system, the body's defense against infectious viruses and bacteria, she said.
Using a mouse model, the researchers identified a gene called Nfil3, which guides the development of the TH17 cells that patrol mucosal surfaces like the intestinal lining and protect against bacterial and fungal infections.
"However, if their numbers are not controlled properly, TH17 cells can produce too much friendly fire and lead to inflammatory diseases such as inflammatory bowel disease (IBD), which afflicts about 600,000 Americans each year," Dr. Hooper said.
"We found that Nfil3 regulates TH17 development by controlling the cellular supply of a protein in T cells called Rorγt that directs the cells to develop into TH17 cells. In mice, the amount of Rorγt in T cells changes during the day-night cycle and is higher at noon than at midnight. This fluctuation causes more TH17 cells to develop at noon when the mice are sleeping," she said.
Mice are nocturnal, meaning their sleep-wake times are the opposite of those in humans.
"When we disrupted the normal day-night light cycles of mice, essentially giving them jet lag, we found that too many TH17 cells developed and accumulated in the intestines. As a result, these mice were more prone to develop an IBD-like disease, due to friendly fire from the overabundance of those inflammatory TH17 cells," she said, adding that it took more than a single day's disruption to change the TH17 concentrations.
Dr. Hooper stressed that it is too soon to tell if the same thing is happening in people, but the possibility is worth studying.
The researchers point out that modern life often involves chronic circadian disruptions, such as night-shift work or jet lag, that other research studies have linked to human inflammatory disease.
"Our findings suggest that the pathologic consequences of circadian disruption may be due in part to direct interactions between the circadian clock and the pathways that regulate proinflammatory immune cell development," the researchers conclude.
###
Others UT Southwestern researchers involved include Dr. Carla Green, Professor of Neuroscience, and Jeremy Stubblefield, a Neuroscience student in the UT Southwestern Graduate School of Biomedical Sciences. Funding was provided by the National Institutes of Health, the Burroughs Wellcome Foundation, and the Howard Hughes Medical Institute.
About UT Southwestern Medical Center
UT Southwestern, one of the premier academic medical centers in the nation, integrates pioneering biomedical research with exceptional clinical care and education. The institution's faculty includes many distinguished members, including five who have been awarded Nobel Prizes since 1985. Numbering more than 2,700, the faculty is responsible for groundbreaking medical advances and is committed to translating science-driven research quickly to new clinical treatments. UT Southwestern physicians provide medical care in 40 specialties to nearly 90,000 hospitalized patients and oversee more than 1.9 million outpatient visits a year.
This news release is available on our home page at utsouthwestern.edu/home/news/index.html
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UT Southwestern researchers identify how body clock affects inflammation
PUBLIC RELEASE DATE:
7-Nov-2013
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Contact: Deborah Wormser deborah.wormser@utsouthwestern.edu 214-648-3404 UT Southwestern Medical Center
DALLAS Nov. 7, 2013 UT Southwestern Medical Center researchers report that disrupting the light-dark cycle of mice increased their susceptibility to inflammatory disease, indicating that the production of a key immune cell is controlled by the body's circadian clock.
The study published in the Nov. 8 edition of Science identifies a previously hidden pathway by which the body's circadian clock controls the numbers of key inflammatory cells called interleukin-17-producing CD4+ T helper cells (TH17). The work could lead to new ways to rev up the body's immune response to infection or dampen that response in the case of autoimmune diseases in which the body attacks its own tissues, said senior author Dr. Lora Hooper, Professor of Immunology and Microbiology and a Howard Hughes Medical Institute (HHMI) Investigator.
Co-authors include Neuroscience Chair and HHMI Investigator Dr. Joseph Takahashi, whose discovery of the mouse and human clock genes led to a description of a conserved circadian clock mechanism in animals. The lead author is Xiaofei Yu, an Immunology student in the UT Southwestern Graduate School of Biomedical Sciences.
"Virtually all life forms on Earth undergo physiological and behavioral changes on a 24-hour daily, or circadian, cycle in accordance with the changes in natural light. Human beings are no exception. Many of our physiological processes, such as eating and sleeping, vary dramatically between day and night. Such processes are controlled by a group of proteins, collectively termed the 'circadian clock,' which function together in individual cells, capturing light cues from the visual and nervous systems and using these cues to regulate gene expression," explained Dr. Hooper, who holds appointments in the Center for the Genetics of Host Defense and the Cancer Immunobiology Center.
Although the circadian clock is known to regulate metabolism and sleep-wake cycles, little was known about whether the circadian clock also regulates the immune system, the body's defense against infectious viruses and bacteria, she said.
Using a mouse model, the researchers identified a gene called Nfil3, which guides the development of the TH17 cells that patrol mucosal surfaces like the intestinal lining and protect against bacterial and fungal infections.
"However, if their numbers are not controlled properly, TH17 cells can produce too much friendly fire and lead to inflammatory diseases such as inflammatory bowel disease (IBD), which afflicts about 600,000 Americans each year," Dr. Hooper said.
"We found that Nfil3 regulates TH17 development by controlling the cellular supply of a protein in T cells called Rorγt that directs the cells to develop into TH17 cells. In mice, the amount of Rorγt in T cells changes during the day-night cycle and is higher at noon than at midnight. This fluctuation causes more TH17 cells to develop at noon when the mice are sleeping," she said.
Mice are nocturnal, meaning their sleep-wake times are the opposite of those in humans.
"When we disrupted the normal day-night light cycles of mice, essentially giving them jet lag, we found that too many TH17 cells developed and accumulated in the intestines. As a result, these mice were more prone to develop an IBD-like disease, due to friendly fire from the overabundance of those inflammatory TH17 cells," she said, adding that it took more than a single day's disruption to change the TH17 concentrations.
Dr. Hooper stressed that it is too soon to tell if the same thing is happening in people, but the possibility is worth studying.
The researchers point out that modern life often involves chronic circadian disruptions, such as night-shift work or jet lag, that other research studies have linked to human inflammatory disease.
"Our findings suggest that the pathologic consequences of circadian disruption may be due in part to direct interactions between the circadian clock and the pathways that regulate proinflammatory immune cell development," the researchers conclude.
###
Others UT Southwestern researchers involved include Dr. Carla Green, Professor of Neuroscience, and Jeremy Stubblefield, a Neuroscience student in the UT Southwestern Graduate School of Biomedical Sciences. Funding was provided by the National Institutes of Health, the Burroughs Wellcome Foundation, and the Howard Hughes Medical Institute.
About UT Southwestern Medical Center
UT Southwestern, one of the premier academic medical centers in the nation, integrates pioneering biomedical research with exceptional clinical care and education. The institution's faculty includes many distinguished members, including five who have been awarded Nobel Prizes since 1985. Numbering more than 2,700, the faculty is responsible for groundbreaking medical advances and is committed to translating science-driven research quickly to new clinical treatments. UT Southwestern physicians provide medical care in 40 specialties to nearly 90,000 hospitalized patients and oversee more than 1.9 million outpatient visits a year.
This news release is available on our home page at utsouthwestern.edu/home/news/index.html
To automatically receive news releases from UT Southwestern via email, subscribe at utsouthwestern.edu/receivenews
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Lyoto Machida made a huge impact in the middleweight division with his first-round knockout over Mark Munoz at UFC Fight Night 30 in London. "The Dragon" returns to the Octagon on Feb. 8 in Jaragua do Sul, Brazil, against Gegard Mousasi, and he needs a win to keep chasing the middleweight title.
"Mousasi is a tough fighter, I’ve seen his fights before and he won titles in other promotions, but I will only focus on his game in my last four or five weeks of camp," Machida said during a Q&A with the fans in Goiania, Brazil on Wednesday.
"He is right below me in the UFC rankings and a win over him would make me achieve even more in this division. But if I lose this fight, it would be complicated. I would fall from fifth place to hell [laughs]," Machida said.
Machida believes he could earn a shot at the middleweight title with a win over Mousasi, a former DREAM and Strikeforce champion who returns to the middleweight division after going 7-1-1 as a light-heavyweight.
"I'm ready (to fight for the title) already, but I have to follow the rankings," he said. "I don’t like to rush things. The right moment will come. I want to keep fighting because it's important for me to keep this rhythm. I want to feel well in this division, this is my place."
Anderson Silva, Machida’s teammate, fights Chris Weidman on Dec. 28 for the middleweight belt at UFC 168, and "The Dragon" doesn’t plan to fight another friend inside the Octagon.
"He said he would never fight me, that we are like brothers," Machida said. "Anderson told me he has other goals, that he was the champion for a long time and he's focused on other goals now, like superfights. He said he would even leave the title to not fight me.
"But we’ll see what happens. I still have to fight Gegard Mousasi in Jaragua do Sul, in February, and I want to think on this fight first. One step at a time."
Country music stars were all dressed up with somewhere to go: the Country Music Awards.
The event in Nashville, Tenn., on Wednesday night was another chance for Taylor Swift to be belle of the ball and this time she did it in a bateau-neck red gown with embroidery and a thin leather belt by Elie Saab.
But there were other stars who took this moment in the spotlight to shine — in sequins and baubles — including Jennifer Nettles, who wore a crystal-and-mirror beaded gown by Naeem Khan on the red carpet and a Georges Chakra blush-colored beaded jumpsuit onstage.
Carrie Underwood did several outfit changes. Among them: She wore a midnight-blue gown with white porcelain cherry blossoms by Theia and a short, black tuxedo-style romper by Chagoury decorated with Swarovski crystals.
Kacey Musgraves went with a short-hemline head turner, choosing a yellow handkerchief dress by Sally LaPointe for her performance. Earlier she was in a Blumarine outfit with a short dress under a long, sheer floral overlay. Kimberly Perry of The Band Perry did the short-long switch, too, in a 14-karat gold and sterling mesh mini by Rubin Singer when she was singing and a more delicate ivory-colored embroidered gown for the arrivals line.
Connie Britton did her own two-in-one turn in a Georges Hobeika gown that had a demure black-beaded collar and a slim, sexy bodice.
___
Follow Samantha Critchell and AP Fashion coverage on Twitter at @AP_Fashion and @Sam_Critchell.
GENEVA (AP) — Officials say U.S. Secretary of State John Kerry will fly to Geneva on Friday to participate in nuclear negotiations with Iran and other major powers.
The officials say Kerry will travel to the talks after a brief stop in Israel, where he will hold a third meeting with Israeli Prime Minister Benjamin Netanyahu. Israel's intense interest in Iran's nuclear ambitions is a likely topic between Netanyahu and Kerry as well as Israeli-Palestinian peace.
Iran's plan to cap some of the country's atomic activities in exchange for selective relief from crippling economic sanctions has been accepted by six world powers, the country's chief nuclear negotiator said Thursday.
Kerry's last-minute decision to join the talks suggests a deal could be imminent.
The officials spoke on condition of anonymity because Kerry has not been formally invited by the Europeans to join the talks.
NIH funds researchers using light to control and monitor neural activity
PUBLIC RELEASE DATE:
7-Nov-2013
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Contact: Traci Peterson tpeterso@uta.edu 817-521-5494 University of Texas at Arlington
University of Texas at Arlington researchers are exploring a better method for initiating certain gene therapies that could better fight the sight-deteriorating disease retinitis pigmentosa.
The National Institutes of Health is funding the research.
Samarendra Mohanty, assistant professor of physics, expects to receive a total of $384,269 over the next two years from the NIH's National Institute of Neurological Disorders and Stroke. His work involves using a near-infrared ultrafast laser beam to deliver genes that allow expression of light-sensitive proteins, called opsins, in specific cells. That proteins' expression allows researchers to influence neural activity through optical or light stimulation a technique known as optogenetics.
In the past, the genes have been delivered to cells by virus. That method can have drawbacks, such as immune responses, in addition to the benefits. In Mohanty's method, a laser beam creates a transient sub-micrometer size hole, which allows for the gene encoding the proteins to permeate through the cell membrane. It can limit the risk of immune response, as well as delivering larger genes than viral methods, he said.
Digant Dave, UT Arlington associate professor of bioengineering at UT Arlington, is the co-investigator on the new grant.
"Our minimally invasive near-infrared method can deliver DNA and other impermeable molecules effectively where you want it and only where you want it," said Mohanty. "For example, in retinitis pigmentosa, only peripheral retina begins to lose light sensitivity due to loss of photoreceptors. This is where a laser can deliver the genes, making those neurons respond to light again. With a virus, the genes will be delivered everywhere, causing complications in areas already working fine."
Optogenetic stimulation also holds promise for influencing neurons in the brain. Scientists, including Mohanty's research group, are researching ways it could be used to understand how the brain works or to intervene in case of neurological disorders or to affect behavior.
Ultimately, Mohanty's team has a goal of creating all optical, or light-based, control and monitoring of cell activity. So, in addition to the light-assisted delivery of genes, the researchers also will work on refining methods for stimulating the neural activity using near-infrared and visible light. Some of those methods are described in a recently published paper called "Fiber-optic two-photon optogenetic stimulation," which appeared in the journal Optics Letters.
Mohanty's lab at UT Arlington also will use a method called phase-sensitive interferometry, to monitor the changes in neurons that result from the activation by light. The interferometry method is called "label-free" because unlike fluorescence, it uses the change in behavior of light rays, rather than staining, to track changes at the sub-nanometer level.
"Dr. Mohanty's proven track record of exploration in the area of optogenetics has earned him this support from the National Institutes of Health," said Alex Weiss, chairman of the College of Science's physics department. "We are pleased to see this promising research supported and look forward to seeing the exciting research findings that this support will make possible."
###
The University of Texas at Arlington is a comprehensive institution of more than 33,300 students and more than 2,200 faculty members in the heart of North Texas. It is the second largest school in The University of Texas System. Visit http://www.uta.edu to learn more.
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NIH funds researchers using light to control and monitor neural activity
PUBLIC RELEASE DATE:
7-Nov-2013
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Contact: Traci Peterson tpeterso@uta.edu 817-521-5494 University of Texas at Arlington
University of Texas at Arlington researchers are exploring a better method for initiating certain gene therapies that could better fight the sight-deteriorating disease retinitis pigmentosa.
The National Institutes of Health is funding the research.
Samarendra Mohanty, assistant professor of physics, expects to receive a total of $384,269 over the next two years from the NIH's National Institute of Neurological Disorders and Stroke. His work involves using a near-infrared ultrafast laser beam to deliver genes that allow expression of light-sensitive proteins, called opsins, in specific cells. That proteins' expression allows researchers to influence neural activity through optical or light stimulation a technique known as optogenetics.
In the past, the genes have been delivered to cells by virus. That method can have drawbacks, such as immune responses, in addition to the benefits. In Mohanty's method, a laser beam creates a transient sub-micrometer size hole, which allows for the gene encoding the proteins to permeate through the cell membrane. It can limit the risk of immune response, as well as delivering larger genes than viral methods, he said.
Digant Dave, UT Arlington associate professor of bioengineering at UT Arlington, is the co-investigator on the new grant.
"Our minimally invasive near-infrared method can deliver DNA and other impermeable molecules effectively where you want it and only where you want it," said Mohanty. "For example, in retinitis pigmentosa, only peripheral retina begins to lose light sensitivity due to loss of photoreceptors. This is where a laser can deliver the genes, making those neurons respond to light again. With a virus, the genes will be delivered everywhere, causing complications in areas already working fine."
Optogenetic stimulation also holds promise for influencing neurons in the brain. Scientists, including Mohanty's research group, are researching ways it could be used to understand how the brain works or to intervene in case of neurological disorders or to affect behavior.
Ultimately, Mohanty's team has a goal of creating all optical, or light-based, control and monitoring of cell activity. So, in addition to the light-assisted delivery of genes, the researchers also will work on refining methods for stimulating the neural activity using near-infrared and visible light. Some of those methods are described in a recently published paper called "Fiber-optic two-photon optogenetic stimulation," which appeared in the journal Optics Letters.
Mohanty's lab at UT Arlington also will use a method called phase-sensitive interferometry, to monitor the changes in neurons that result from the activation by light. The interferometry method is called "label-free" because unlike fluorescence, it uses the change in behavior of light rays, rather than staining, to track changes at the sub-nanometer level.
"Dr. Mohanty's proven track record of exploration in the area of optogenetics has earned him this support from the National Institutes of Health," said Alex Weiss, chairman of the College of Science's physics department. "We are pleased to see this promising research supported and look forward to seeing the exciting research findings that this support will make possible."
###
The University of Texas at Arlington is a comprehensive institution of more than 33,300 students and more than 2,200 faculty members in the heart of North Texas. It is the second largest school in The University of Texas System. Visit http://www.uta.edu to learn more.
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AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Police stand outside a building near the scene of a multiple shooting that took place at Al's Place Barber Shop in Detroit, Wednesday, Nov. 6, 2013. Detroit police say gunfire broke out at the barbershop known for gambling activity. Police Chief James Craig told reporters that police were looking for two vehicles that the suspects may have been using, a 2004 white Chevrolet Impala that may have a broken window and bullet holes in the back, and a 2004 black Impala. (AP Photo/Carlos Osorio)
Police stand outside a building near the scene of a multiple shooting that took place at Al's Place Barber Shop in Detroit, Wednesday, Nov. 6, 2013. Detroit police say gunfire broke out at the barbershop known for gambling activity. Police Chief James Craig told reporters that police were looking for two vehicles that the suspects may have been using, a 2004 white Chevrolet Impala that may have a broken window and bullet holes in the back, and a 2004 black Impala. (AP Photo/Carlos Osorio)
Detroit Police Chief James Craig addresses the media during a news conference in Detroit, Thursday, Nov. 7, 2013. Craig said a man has been apprehended and will be questioned about a barbershop shooting that killed three people and wounded several more on Wednesday night on the city's east side. Craig said that the man was wearing body armor when he was arrested on unrelated felony charges in suburban Rochester after the shootings. (AP Photo/Carlos Osorio)
Police stand outside the scene of a multiple shooting at Al's Place Barber Shop in Detroit, Wednesday, Nov. 6, 2013. Detroit police say gunfire broke out at the barbershop known for gambling activity. Police Chief James Craig told reporters that police were looking for two vehicles that the suspects may have been using, a 2004 white Chevrolet Impala that may have a broken window and bullet holes in the back, and a 2004 black Impala. (AP Photo/Carlos Osorio)
Police gather at a scene of a multiple shooting at Al's Place Barber Shop, Wednesday, Nov. 6, 2013, in Detroit. Detroit police say gunfire broke out at the barbershop known for gambling activity, leaving at least three people dead. (AP Photo/Detroit Free Press, Andre J. Jackson) DETROIT NEWS OUT; NO SALES; TV OUT; INTERNET OUT; MANDATORY CREDIT
People stand outside a barber shop, Wednesday, Nov. 6, 2013 in Detroit. Gunfire broke out Wednesday evening at a Detroit barbershop known for gambling activity, leaving at least two men dead, police say. (AP Photo/Detroit News, Elizabeth Conley) DETROIT FREE PRESS OUT; HUFFINGTON POST OUT
DETROIT (AP) — A convicted felon who was wearing body armor when police arrested him in a Detroit suburb will be questioned in an investigation into the fatal shooting of three men in a back gambling room of an east side barbershop.
Detroit Police Chief James Craig described the man as a person of interest in Wednesday evening's shooting at Al's Barber Shop that left six other people wounded. Speaking at a Thursday news conference at police headquarters, Craig said the bloodshed may have stemmed from an ongoing feud.
Craig did not name the person of interest and provided few details about the arrest. He said Rochester police picked up the man north of Detroit without incident on an unrelated felonious assault charge.
"Police wear body armor. Why would a community member be driving around in body armor?" said Craig, who described the latest mass shooting in Detroit as "urban terrorism."
Craig said 20 to 30 people were gambling in a tiny back room at the barbershop in a strip mall shortly before 6 p.m. when shots were fired from a high-powered rifle through an open back door.
When the gunfire began, the gamblers tried to escape the room through a door that led back into the barbershop, but that the jam of bodies prevented them from escaping.
"The shooter struck nine of the individuals inside the location," Craig said, adding that many people in the room were armed and at least one returned fire.
"Officers were able to develop information that there was an ongoing feud between a particular individual and several members of the gambling party," the chief said.
Craig declined to say whether police think the person of interest is the shooter. But he would say that the shooter is suspected in at least two other violent crimes.
Two of Wednesday's shooting victims died inside the gambling room. The third died later at a hospital. The conditions of the survivors were not available.
A witness told police that the shooter pulled into a rear alley and fired shots at someone in a pickup truck. He then got out of the car and began shooting into the open rear door of the barbershop.
Wednesday night police said they were seeking two vehicles believed to have been involved in the shooting.
Lorne Carter told the Detroit Free Press that he was smoking a cigarette against the wall of a nearby business when he heard what sounded like 30 to 40 rapid shots.
Detroit has one of the highest violent crime rates in the country. Including Wednesday night's barbershop slayings, 289 criminal homicides have been committed so far this year in Detroit.
Criminal homicides accounted for 386 of the 461 death investigations in 2012.
"Anytime you have ... shootings of nine, it certainly does rise up as one of Detroit's more violent incidents," Craig said.
Craig said one person was shot and three others wounded during a shooting in October 2012 on the city's west side. Seven teenagers — including five students at Cody Ninth Grade Academy — were shot and wounded while waiting at a bus stop near that school in June 2009.
___
Associated Press writers David N. Goodman and David Runk contributed to this report.
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